Respiratory Trials·org

Topic

Cystic fibrosis

Mucolytics, inhaled antibiotics, and the CFTR modulators.

What the evidence shows

CFTR modulators corrected the basic defect, and the sweat chloride fall in the first positive trial made that literally measurable (STRIVE). Ivacaftor gave a 10-point FEV1 gain but only to the 4% of patients carrying G551D.

Triple therapy extended that to roughly 90% of patients. Elexacaftor-tezacaftor-ivacaftor produced a 14-point improvement in predicted FEV1 and cut exacerbations by 63% in genotypes where earlier modulators had failed (VX17-445-102) — the trial that changed the trajectory of the disease. A once-daily successor is non-inferior on lung function and reduces treatment burden (SKYLINE).

The field's question is now what to stop. In patients on triple modulator therapy with well-preserved lung function, discontinuing hypertonic saline or dornase alfa for six weeks was non-inferior to continuing (SIMPLIFY). Six weeks is short and the participants were well, so this licenses deprescribing in stable patients rather than in everyone.

CFTR modulators

Trial Year Therapy Effect Finding
SKYLINE 2025 Vanzacaftor/tezacaftor/deutivacaftor The once-daily triple matched the twice-daily standard on lung function
VX17-445-102 2019 Elexacaftor/tezacaftor/ivacaftor Triple modulator therapy gave a 14-point FEV1 gain to the 90% of CF patients with a single Phe508del allele
STRIVE 2011 Ivacaftor Ivacaftor produced a 10-point FEV1 gain in G551D cystic fibrosis — the first drug to correct the basic defect

De-escalating older therapies

Trial Year Therapy Effect Finding
SIMPLIFY 2023 Hypertonic saline and dornase alfa withdrawal Patients on triple modulator therapy could stop hypertonic saline or dornase alfa without losing lung function

Key references

Guidelines, standards and reviews for this topic.

Studies not yet summarised

On the reading list for this topic and awaiting a full record. Listed here so nothing is hidden.